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Pathophysiology
Citations 361-370 of 1280 total displayed.
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Past content
(since Jan 1999):
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- Basic Science for Clinicians
Physiological Basis of Clinically Used Coronary Hemodynamic Indices
- Jos A.E. Spaan, Jan J. Piek, Julien I.E. Hoffman, and Maria Siebes
Circulation 2006;113:446-455, doi:10.1161/CIRCULATIONAHA.105.587196
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- Vascular Medicine
Disruption of the Cathepsin K Gene Reduces Atherosclerosis Progression and Induces Plaque Fibrosis but Accelerates Macrophage Foam Cell Formation
- E. Lutgens, S.P.M. Lutgens, B.C.G. Faber, S. Heeneman, M.M.J. Gijbels, M.P.J. de Winther, P. Frederik, I. van der Made, A. Daugherty, A.M. Sijbers, A. Fisher, C.J. Long, P. Saftig, D. Black, M.J.A.P. Daemen, and K.B.J.M. Cleutjens
Circulation 2006;113:98-107; published online before print as doi:10.1161/CIRCULATIONAHA.105.561449
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- Contemporary Reviews in Cardiovascular Medicine
What a Cardiologist Needs to Know About Patients With Human Immunodeficiency Virus Infection
- Priscilla Y. Hsue and David D. Waters
Circulation 2005;112:3947-3957, doi:10.1161/CIRCULATIONAHA.105.546465
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- Articles
Disruption of the Cathepsin K Gene Reduces Atherosclerosis Progression and Induces Plaque Fibrosis but Accelerates Macrophage Foam Cell Formation
- E. Lutgens, S. P.M. Lutgens, B. C.G. Faber, S. Heeneman, M. M.J. Gijbels, M. P.J. de Winther, P. Frederik, I. van der Made, A. Daugherty, A. M. Sijbers, A. Fisher, C. J. Long, P. Saftig, D. Black, M. J.A.P. Daemen, and K. B.J.M. Cleutjens
Circulation published December 19, 2005, doi:10.1161/CIRCULATIONAHA.105.561449
[Abstract]
- Special Reports
Atherosclerosis 2005: Recent Discoveries and Novel Hypotheses
- Pascal J. Goldschmidt-Clermont, Mark A. Creager, Douglas W. Lorsordo, Gregory K.W. Lam, Momtaz Wassef, and Victor J. Dzau
Circulation 2005;112:3348-3353, doi:10.1161/CIRCULATIONAHA.105.577460
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- Epidemiology
Which Hemostatic Markers Add to the Predictive Value of Conventional Risk Factors for Coronary Heart Disease and Ischemic Stroke?: The Caerphilly Study
- Ann Smith, Chris Patterson, John Yarnell, Ann Rumley, Yoav Ben-Shlomo, and Gordon Lowe
Circulation 2005;112:3080-3087, doi:10.1161/CIRCULATIONAHA.105.557132
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- Vascular Medicine
The Thromboxane A2 Receptor Antagonist S18886 Prevents Enhanced Atherogenesis Caused by Diabetes Mellitus
- Adriana Zuccollo, Chaomei Shi, Roberto Mastroianni, Karlene A. Maitland-Toolan, Robert M. Weisbrod, Mengwei Zang, Shanqin Xu, Bingbing Jiang, Jennifer M. Oliver-Krasinski, Antonio J. Cayatte, Stefano Corda, Gilbert Lavielle, Tony J. Verbeuren, and Richard A. Cohen
Circulation 2005;112:3001-3008; published online before print as doi:10.1161/CIRCULATIONAHA.105.581892
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- Vascular Medicine
Involvement of Thromboxane Receptor in the Proatherogenic Effect of Isoprostane F2 -III: Evidence From Apolipoprotein E and LDL ReceptorDeficient Mice
- Myan Tang, Tillmann Cyrus, Yuemang Yao, Luigina Vocun, and Domenico Praticò
Circulation 2005;112:2867-2874, doi:10.1161/CIRCULATIONAHA105.562223
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- Vascular Medicine
Sevelamer Prevents Uremia-Enhanced Atherosclerosis Progression in Apolipoprotein EDeficient Mice
- Olivier Phan, Ognen Ivanovski, Thao Nguyen-Khoa, Nadya Mothu, Jesus Angulo, Ralf Westenfeld, Markus Ketteler, Natalie Meert, Julien Maizel, Igor G. Nikolov, Raymond Vanholder, Bernard Lacour, Tilman B. Drüeke, and Ziad A. Massy
Circulation 2005;112:2875-2882, doi:10.1161/CIRCULATIONAHA105.541854
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- Articles
The Thromboxane A2 Receptor Antagonist S18886 Prevents Enhanced Atherogenesis Caused by Diabetes Mellitus
- Adriana Zuccollo, Chaomei Shi, Roberto Mastroianni, Karlene A. Maitland-Toolan, Robert M. Weisbrod, Mengwei Zang, Shanqin Xu, Bingbing Jiang, Jennifer M. Oliver-Krasinski, Antonio J. Cayatte, Stefano Corda, Gilbert Lavielle, Tony J. Verbeuren, and Richard A. Cohen
Circulation published October 31, 2005, doi:10.1161/CIRCULATIONAHA.105.581892
[Abstract]
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