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(Circulation. 1959;20:190.)
© 1959 American Heart Association, Inc.

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Circulatory Changes in Acute Glomerulonephritis

VALENTINO DEFAZIO M.D.¹; RAYMOND C. CHRISTENSEN M.D.¹; TIMOTHY J. REGAN M.D.¹; LESEM J. BAER M.D.¹; YOSHIKAZU MORITA M.D.¹; HARPER K. HELLEMS M.D.¹

¹ From the Cardiovascular Research Laboratories Departments of Medicine, Wayne State University College of Medicine, City of Detroit Receiving Hospital, and Dearborn Veterans Administration Hospital, Detroit, Mich.

Conflicting opinions have been expressed as to the mechanism of pulmonary and peripheral congestion frequently observed in the oliguric stage of acute glomerulonephritis. Hemodynamic studies in 7 patients studied during the oliguric edematous phase of the disease demonstrated that these patients have high ventricular filling pressures associated with an increase in cardiac output and are, therefore, examples of a high output congestive state.

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