Donate Help Contact The AHA Sign In Home
American Heart Association
Circulation
Search: search_blue_button Advanced Search
Circulation. 1961;24:250-262

This Article
Right arrow Full Text (PDF)
Right arrow Alert me when this article is cited
Right arrow Alert me if a correction is posted
Services
Right arrow Email this article to a friend
Right arrow Similar articles in this journal
Right arrow Similar articles in PubMed
Right arrow Alert me to new issues of the journal
Right arrow Download to citation manager
Right arrow Request Permissions
Citing Articles
Right arrow Citing Articles via HighWire
Right arrow Citing Articles via Google Scholar
Google Scholar
Right arrow Articles by MARCH, H. W.
Right arrow Articles by HULTGREN, H. N.
Right arrow Search for Related Content
PubMed
Right arrow PubMed Citation
Right arrow Articles by MARCH, H. W.
Right arrow Articles by HULTGREN, H. N.

(Circulation. 1961;24:250.)
© 1961 American Heart Association, Inc.


The Reopened Ventricular Septal Defect

A Syndrome Following Unsuccessful Closure of Interventricular Septal Defects Particularly in Association with Infundibular Stenosis

HAROLD W. MARCH M.D.1; FRANK GERBODE M.D.1; HERBERT N. HULTGREN M.D.1

1 From the Cardiology Laboratory, Stanford Medical Center, Palo Alto, and the San Francisco Institute of Medical Sciences, Presbyterian Medical Center, San Francisco, California.

The persistence of a left-to-right shunt due to the unsuccessful repair of a ventricular septal defect associated with infundibular stenosis has been described.

A characteristic clinical picture has been reported. The main features include congestive heart failure, tricuspid insufficiency, and increased pulmonary blood flow.

The severity of the symptoms appears to be due to the persistence of a left-to-right shunt at a time when the myocardium has been affected adversely by such operative insults as potassium arrest and ventriculotomy.

The particular difficulties attending the repair of a ventricular septal defect with infundibular stenosis include the proximity of the defect to the hypertrophied muscle mass, the soft muscular margin of such a defect, and the tension on the suture line. Ivalon and Teflon prostheses have been disappointing in their failure to maintain the integrity of the repair.

All of the patients were reoperated upon. Five survived and their defects are now closed. The second repairs were abetted by the development of a fibrous scar around the margin of the defect. The five survivors are in good health and there is no evidence of a remaining shunt.

As a consequence of these experiences, surgical technics were modified. The defects are now closed by direct suture with interrupted silk, followed by the application of a crimped Dacron patch over the repair. The results have been encouraging and since the new technic has been adopted, there have been virtually no recurrences.




This article has been cited by other articles:


Home page
ANGIOLOGYHome page
B. Y. Dy, V. Maranhao, Sing San Yang, S. G.G. Ablaza, H. Goldberg, and H. Goldberg
Clinical and Physiologic Evaluation of Residual Ventricular Septal Defects
Angiology, April 1, 1969; 20(4): 207 - 212.
[PDF]