Circulation, Vol 57, 111-115, Copyright © 1978 by American Heart Association
ME Josephson, LN Horowitz and JA Kastor
The mechanism of supraventricular tachycardia was evaluated in twelve
patients with mitral valve prolapse utilizing standard intracardiac
recording and stimulation techniques. Eight patients had normal
electrocardiograms, three had a short PR interval and normal QRS (Lown-
Ganong-Levine syndrome) and one had Wolff-Parkinson-White type A. Six of
the eight patients with normal electrocardiograms were demonstrated to have
atrioventricular bypass tracts. Five patients had A-V nodal re- entrant
supraventricular tachycardia. In two patients the bypass tract could be
demonstrated to conduct antegradely when the left atrium was paced via the
coronary sinus, while in four the bypass tract only conducted retrogradely.
In all seven patients with atrioventricular bypass tracts, the accessory
pathway was left-sided. We conclude that a) supraventricular tachycardia in
the mitral valve prolapse syndrome appears related to a high frequency of
bypass tracts; b) electrophysiological studies are required to diagnose
these bypass tracts; and c) the atrioventricular bypass tracts may be
related to the mitral valve abnormality since they are always left-sided.
ARTICLES
Paroxysmal supraventricular tachycardia in patients with mitral valve prolapse
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L. Gould and C. Gopalaswamy Mitral Valve Prolapse Angiology, May 1, 1984; 35(5): 290 - 299. [PDF] |
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