Circulation, Vol 86, 699-702, Copyright © 1992 by American Heart Association
AE Taylor, DC Johnson and H Kazemi
Although the number of cardiovascular deaths associated with environmental
tobacco smoke cannot be predicted with absolute certainty, the available
evidence indicates that environmental tobacco smoke increases the risk of
heart disease. The effects of environmental tobacco smoke on cardiovascular
function, platelet function, neutrophil function, and plaque formation are
the probable mechanisms leading to heart disease. The risk of death due to
heart disease is increased by about 30% among those exposed to
environmental tobacco smoke at home and could be much higher in those
exposed at the workplace, where higher levels of environmental tobacco
smoke may be present. Even though considerable uncertainty is a part of any
analysis on the health affects of environmental tobacco smoke because of
the difficulty of conducting long-term studies and selecting sample
populations, an estimated 35,000-40,000 cardiovascular disease-related
deaths and 3,000- 5,000 lung cancer deaths due to environmental tobacco
smoke exposure have been predicted to occur each year. The AHA's Council on
Cardiopulmonary and Critical Care has concluded that environmental tobacco
smoke is a major preventable cause of cardiovascular disease and death. The
council strongly supports efforts to eliminate all exposure of nonsmokers
to environmental tobacco smoke. This requires that environmental tobacco
smoke be treated as an environmental toxin, and ways to protect workers and
the public from this health hazard should be developed. According to a 1989
Gallup survey commissioned by the American Lung Association, 86% of
nonsmokers think that environmental tobacco smoke is harmful and 77%
believe that smokers should abstain in the presence of nonsmokers.(ABSTRACT
TRUNCATED AT 250 WORDS)
ARTICLES
Environmental tobacco smoke and cardiovascular disease. A position paper from the Council on Cardiopulmonary and Critical Care, American Heart Association
Office of Scientific Affairs, American Heart Association, Dallas, TX 75231-4596.
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