Circulation, Vol 90, 272-281, Copyright © 1994 by American Heart Association
HR Grogin, RJ Lee, M Kwasman, LM Epstein, DJ Schamp, MD Lesh and MM Scheinman
BACKGROUND: Several mechanisms have been proposed to explain the
pathogenesis of tachycardia in patients with Mahaim tracts. The tachycardia
may involve antegrade conduction over an atriofascicular pathway with
decremental properties or a nodofascicular pathway. METHODS AND RESULTS: We
report six patients with recurrent episodes of preexcited tachycardia with
findings consistent with "Mahaim tract" conduction. All patients exhibited
decremental antegrade preexcited conduction with atrial pacing and a
preexcited tachycardia with initial activation of the proximal right bundle
branch. In four patients (group 1), atrial premature complexes (APCs)
induced at the tricuspid annulus just after the inscription of the septal
atrial electrogram and during left bundle branch block preexcited
tachycardia advanced the next preexcited ventricular complex. In these
patients, discrete Mahaim potentials were inscribed over the right
anterolateral or lateral tricuspid annulus. Two patients (group 2) had
evidence of dual atrioventricular nodal conduction. APCs during left bundle
branch block tachycardia just after inscription of the septal atrial
electrogram failed to advance the next ventricular complex with similar
preexcited morphology, and no Mahaim potentials could be recorded from the
tricuspid annulus. In group 1 patients, application of radiofrequency
energy to sites recording the Mahaim potentials resulted in tachycardia
cure. For patients in group 2, selective slow atrioventricular nodal
pathway ablation in the midseptal region resulted in complete ablation of
both the slow atrioventricular nodal pathway and Mahaim conduction in two
patients. CONCLUSIONS: Mahaim tachycardia can be due to atriofascicular
pathways, which may be ablated over the right tricuspid annulus, or to
septal pathways, which may arise from the slow atrioventricular nodal
pathway in patients with dual atrioventricular nodal physiology. In the
latter circumstance, successful ablation is achieved by placing the lesion
in the midseptal region.
ARTICLES
Radiofrequency catheter ablation of atriofascicular and nodoventricular Mahaim tracts
Cardiovascular Research Institute, University of California San Francisco.
This article has been cited by other articles:
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M. Haghjoo, A. Arya, Z. Emkanjoo, and M. A. Sadr-Ameli Is the activation potential of Mahaim pathway always a fast potential? Implication for radiofrequency catheter ablation Europace, January 1, 2005; 7(5): 440 - 446. [Abstract] [Full Text] [PDF] |
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