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Circulation. 2008;118:e504
doi: 10.1161/CIRCULATIONAHA.107.736512
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(Circulation. 2008;118:e504.)
© 2008 American Heart Association, Inc.


Images in Cardiovascular Medicine

Eustachian Valve Thrombosis

Javier Escota-Villanueva, MD; Isaac Lacambra-Blasco, MD; Carmen Gonzalvo, MD

From the Echocardiography Unit of Cardiology Medical Service (J.E.-V.), Echocardiography Unit of Cardiology Medical Service (I.L.-B.), and Department of Internal Medicine (C.G.), Hospital Clínico Universitario "Lozano Blesa," Zaragoza, Spain.

A 61-year-old man who presented with syncope for several minutes recovered spontaneously. On admission, the patient was asymptomatic, and the initial physical examination was normal. The first complementary tests (ECG, blood laboratory, chest radiography, cerebral computed tomography, and pulmonary scintigraphy) showed no abnormalities.

A transthoracic echocardiogram showed a worm-like mass in the right atrium not clearly attached to the heart wall. Unfractionated heparin was initiated with the initial suspicion of intracardiac thrombus. Thirty-six hours later, a transesophageal echocardiography revealed a 4x8-mm mobile mass on the eustachian valve (Movie I), the size of which was decreased considerably with respect to the previous study after an intensive anticoagulation therapy. This finding supported the initial diagnosis of intracardiac thrombosis, and oral anticoagulation with warfarin was begun. After discharge (3 and 6 months later), 2 new transesophageal studies were made that provided evidence of the complete disappearance of the eustachian valve thrombus (Movies II and III).

The eustachian valve in adult is an embryological remainder of the fetal venous sinus valve. This structure is located in the right atrium and is a common, not pathological, echocardiographic finding. It is known that in 4% to 18% of pulmonary embolisms, thrombi are discovered in the right side of the heart; however, their presence on the eustachian valve is very unusual. Only 3 cases have been reported with this finding: 2 patients in the context of pulmonary embolism and 1 who was asymptomatic. In the 3 cases, an aggressive treatment (fibrinolytic therapy or systemic anticoagulation) achieved complete thrombus lysis.


*    Acknowledgments
 
Disclosures

None.


*    Footnotes
 
The online Data Supplement is available with this article at http://circ.ahajournals.org/cgi/content/full/118/13/e504/DC1.

Reprint requests to Javier Escota-Villanueva, C/Desiderio Escosura, No. 42, Entlo. 1, 50005 Zaragoza, Spain. E-mail jaescota@yahoo.es





This Article
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Right arrow Data Supplement
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Citing Articles
Right arrow Citing Articles via Google Scholar
Google Scholar
Right arrow Articles by Escota-Villanueva, J.
Right arrow Articles by Gonzalvo, C.
Right arrow Search for Related Content
PubMed
Right arrow PubMed Citation
Right arrow Articles by Escota-Villanueva, J.
Right arrow Articles by Gonzalvo, C.
Related Collections
Right arrow Arterial thrombosis
Right arrow Heparin
Right arrow Coumarins
Right arrow Echocardiography