(Circulation. 1998;98:2095-2097.)
© 1998 American Heart Association, Inc.
Images in Cardiovascular Medicine |
From the Cardiology Division and Division of Cardiovascular Surgery (B.-C.C.), Cardiovascular Center, Yonsei University, Seoul, Korea.
Correspondence to Jong-Won Ha, MD, Cardiology Division, Cardiovascular Center, Yonsei University College of Medicine, CPO Box 8044, Seoul, Korea. E-mail jwha{at}yumc.yonsei.ac.kr
Percutaneous mitral balloon valvotomy (PMV) has evolved
into an effective method for the treatment of patients with
symptomatic mitral stenosis. An increase in mitral
regurgitation can occur in
45% of patients
undergoing PMV. Severe mitral regurgitation can be
caused by rupture of chordae or of a papillary muscle. Noncommissural
tearing of the mitral leaflet is also an important mechanism of severe
regurgitation after PMV.
A 35-year-old woman presented with exertional dyspnea that had
been present for 3 months. Physical examination revealed a
chronically ill-looking appearance with malar flush and accentuated
first heart sound, opening snap, and diastolic rumble at
the apex. The ECG revealed normal sinus rhythm with left atrial
enlargement. Transthoracic
echocardiography revealed severe mitral
stenosis with trivial mitral regurgitation. The
echo score according to Wilkins et al was
8 (mobility, 2;
thickening, 2; subvalvular, 2; and calcification, 2). Balloon
mitral valvotomy was performed with a 28-mm Inoue balloon catheter. The
effective balloon dilating area was
6.52
m2. After 1 dilatation, the patient
complained of chest tightness and dyspnea, with a markedly elevated
v wave in the left atrial pressure tracing.
Transesophageal echocardiography
revealed severe eccentric mitral regurgitation toward
the anterior wall of the left atrium, with suspicious tearing of the
posterior mitral leaflet. These findings were confirmed at subsequent
mitral valve replacement surgery with a prosthetic valve. The
patient subsequently recovered and was uneventfully discharged days
later.
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Footnotes
The editor of Images in Cardiovascular Medicine is Hugh A. McAllister, Jr, MD, Chief, Department of Pathology, St Luke's Episcopal Hospital and Texas Heart Institute, and Clinical Professor of Pathology, University of Texas Medical School and Baylor College of Medicine.
Circulation encourages readers to submit cardiovascular images to Dr Hugh A. McAllister, Jr, St Luke's Episcopal Hospital and Texas Heart Institute, 6720 Bertner Ave, MC1-267, Houston, TX 77030.
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