(Circulation. 2000;102:1337.)
© 2000 American Heart Association, Inc.
Images in Cardiovascular Medicine |
From the Mayo Clinic, Rochester, Minn.
Correspondence and reprint requests to Guy S. Reeder, MD, Division of Cardiovascular Diseases, Mayo Clinic, 200 First Street SW, Rochester, MN 55905. E-mail reeder.guy@mayo.edu
A56-year-old woman presented for an evaluation of
a tremor. In the course of her evaluation, her serum potassium was
found to be 2.5 meq/L (normal range, 3.6 to 4.8 meq/L); this was
probably secondary to diuretic therapy. She was subsequently
admitted to the hospital for potassium replacement and further
evaluation of the tremor. Physical examination was remarkable only for
a high-amplitude left upper extremity tremor. The patients blood
pressure was 120/70 mm Hg. An ECG (Figure 1
) suggested ventricular
tachycardia, and the patient was transferred to the
coronary care unit for further observation. In the unit, the
patient was hemodynamically stable and had no
complaints. Her pulse was 72 bpm and regular. When a repeat ECG was
performed (Figure 2
), the possibility of
a tremor-induced artifact was raised. This was confirmed as the cause
of the ECG findings when a third ECG (Figure 3
) was performed while holding the
patients left upper extremity.
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The possibility of artifact as a cause of ECG findings should always be
considered in an otherwise asymptomatic patient who is
hemodynamically stable. In our patient, a cursory
overview of the ECG suggested ventricular
tachycardia. However, close scrutiny, particularly of lead
V2 on Figure 2
, clearly shows the QRS complexes buried in the
wide amplitude, repetitive electrical activity. Tremor-induced ECG
artifact should always be
This article has been cited by other articles:
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